Hemostaza pierwotna pdf free. Information of the substance preparation and of the company. The most comprehensive portfolio of hemostasis solutions. HEMOSTAZA . DZIECI: profilaktyka pierwotna; obecnie do 18 r.ż. starania PSCH o przedłużenie do 26 r.ź. leki osoczopochodne /.

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So, let me erase everything. The way we do that is with this family of proteins called coagulation factors. Charakterystyka epidemiologicznokliniczna boreliozy z lyme w regionie polnocnowschodnim polski. But what thrombomodulin will do is if there’s thrombin floating around in our blood and we want to make sure that we don’t clot too much, is it’ll get thrombin and it’ll change what thrombin actually normally does, which is make a clot.

Here, we have our blood vessel and it’s been damaged. Which again, in this case we don’t need because there’s no injury, and so we don’t need to make the clot. And both of these are provided by the platelets. The second way that we can activate the coagulation cascade is through another protein that is secreted by the endothelial cells whenever we have damage to the blood vessels from outside of our bodies.


Calcium is released during primary hemostasis when platelets get activated. There are two ways that we can activate the coagulation cascade.

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Pozar microwave engineering e books electrical engineering. So this is the balance between hemostasis and anticoagulation and thrombolysis. And the way that our bodies do this is through two processes, called anticoagulation, and thrombolysis.

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Hemostaza pierwotna pdf files

Podstawy diagnozowania i leczenia chorob nowotworowych u. Fibrin doesn’t flow in our blood as fibrin, it carries around an extra piece of protein, and when it has this extra piece of protein, it’s called fibrinogen. This protein is called tissue factor or it’s also called thromboplastin.

And now in anticoagulation, we have anti-thrombin III, and we want to prevent a clot. In order to prevent it from getting loose, you double-knot it or triple-knot it. They’re similar, but there are different kinds. The cells that communicate with the blood. This is the same distance as if you were to travel around the Earth a little bit less than four times.

This is the first step of stopping any bleeding. And that happens during secondary hemostasis. And the way we do this is through hemostasis. There’s no injured endothelial cells, so we don’t need hemostasis to happen, we don’t need platelets to get there. The way that it helps with anticoagulation is that it inactivates two specific coagulation factors. Hamilton1,6, 1departmentof anatomyand cell biology, schulich schoolof medicineand dentistry, theuniversity of westernontario, richmond.


This begins immediately after there’s been an injury. The way we hemostxza this is through hemostasis. So let me erase this now. And we want to make sure that we stop any bleeding or minimize it. So now let’s zoom out and take a look at the entire picture of hemstaza anticoagulation and thrombolysis works. So the way that we take care of that is through anticoagulation and thrombolysis.

So even though hemostasis is good, if we have too much clotting, then that can be bad. But plasmin isn’t floating around in our blood all the time, or else we wouldn’t be able to clot at all, because plasmin would be breaking down fibrin and fibrinogen all the time.