A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.

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Espectrophotometric solid-phase tissue plasminogen activator activity assay Sofia-tPA for high-fibrin-affinity tissue plasminogen activators. Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Disorders of hemostasis in childhood: The Kinston antiphospholipid group.

Are you a health professional able to prescribe or dispense drugs? Results Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Antithrombin heparin cofactor assay with new chromogenic substrates.

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¿Trombofilia y enfermedad de Perthes? | Anales de Pediatría

Thromb Haemost, 62pp. Blood, 80pp. Patients and method Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients. Thromb Haemost, 69pp.

Genetics aspects of Perthes disease: Identification of the same factor V gene mutation in 47 of 50 thrombosis-prone families with inherited resistance to activated protein C. Lancet,pp.

Familial hypofibrinolysis and venous thrombosis. Anal Biochem,pp. Clin Chem, 32pp.

Síndrome de Legg-Calvé-Perthes

J Clin Invest, 94pp. Association of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease. Si continua navegando, consideramos que acepta su uso. Familial idiopathic oeteonecrosis mediated by familial hypofibrinolysis witn high levels of plasminogen activator inhibitor. Am J Clin Pathol, 94pp.

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Clin Orthop,pp. Changes in coagulation-fibrinolysis system in Legg-Perthes disease: Determination of plasminogen activator and its fast inhibitor in plasma. Conclusions Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe d underlying cause of avascular necrosis of the hipjoint. Anticoagulant protein C pathway defective in majority of thromboembolic patients.


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You can change the settings or obtain more information by clicking here. The pdiatria international anticardiolipin standardization workshop. It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis.

Br J Hematol, 71pp.

The remaining patients were considered withinthe normal range when age was taken into account. Clin Othop,pp. Resistance to activated protein C and Legg-Perthes disease. Blood collection in strongly acid citrate anticoagulant used in a study of dietary influence of basal Tpa activity.

Blood, 82pp. Aguirre Canyadell aI.